Mice homozygous for the obese spontaneous mutation, (Lepob commonly

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Recently, we found that ob/ob mice have defective liver catabolism of HDL

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hyperglycaemia and improve glucose homeostasis in insulin-resistant ob/ob

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Reduced liver damage in ob/ob compared with +/? mice after administration of

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Male and female heterozygous (OB/ob) mice on a C57BL/6 background were

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(ob/ob), these mice develop severe type 2 diabetes (fasting glucose .400 mg/dL).

glucose, ob/ob islets showed an 8-fold greater absolute increase in insulin

Objective: We studied ob/ob and wild-type (WT) mice to characterize the adipose

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Improved glycaemic control in obese diabetic ob/ob mice using. N-terminally

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May 29, 2007 . The physiology of obese-hyperglycemic mice [ob/ob mice]. Lindström P.

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This study was aimed at characterization of PDN in leptin-deficient (ob/ob) mice,

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Obese mice (ob/ob) are phenotypically distinguished from their lean littermates (

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The ob/ob or obese mouse is a mutant mouse that eats excessively and becomes

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When ob/ob mice are treated with injections of leptin they lose their excess fat

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