Cell fate mapping by using Sox9-Cre;ROSA26 reporter (R26R) mice revealed

Cre expression removed the mRFP1 gene, allowing expression of Sox9 and

Oct 11, 2005 . In the developing skeleton, Sox9 was expressed before Runx2, an early

Abbreviations: SOX9, SRY-related HMG box transcription factor 9; Cre,

May 2, 2011 . 4.1 Targeted reduction of Sox9 in heart valves . . . . . . . . . . . . . 41. 4.2

Jan 7, 2010 . Methods and Results: Using histological and molecular analyses, we report that,

Sep 6, 2002 . The Sox9flox heterozygotes that harborPrx1–Cre are viable and fertile .

Sox9 conditional null allele. • Sox9 flox allele (Chaboissier and Schedl). •

Mice bearing Sox9flox/flox [20] and AMH-Cre transgenes [21] were crossed to

Oct 11, 2005 . In the developing skeleton, Sox9 was expressed before Runx2, an early osteoblast

May 16, 2011 . When Sox9 is knocked out in just chondrocytes using a cre-loxP strategy where

In the absence of T, the Cre-ER(T) fusion protein is retained in the cytoplasm and

Feb 15, 2009 . Generation of Taceflox/flox mice and Sox9-Cre knock-in mice was previously

View Sox9 allele: origin, molecular description, curated phenotype details, gene

We then created conditional knockout mice of Rbpj in chondroprogenitor cells (

Generation of Taceflox/flox mice and Sox9-Cre knock-in mice was previ- .

There was a significant difference in the number of primary neurosphere between

Tg(Sox9-cre/ERT2)1Msan transgene insertion 1, Maike Sander Gene

View Sox9 allele: origin, molecular description, curated phenotype details, gene

View Tg(Sox9-cre/ERT2)1Msan allele: origin, molecular description, gene

Feb 15, 2009 . Generation of Taceflox/flox mice and Sox9-Cre knock-in mice was previously

functions in skeletal development and the formation of other organs, we have

Cre expression removed the mRFP1 gene, allowing expression of Sox9 and

Here we analyzed spinal cord development of mice in which the transcription

Apr 11, 2011. mesenchyme with Prx1-Cre, in chondro-osteoprogenitors with Sox9-Cre and in

generated a transgenic mouse line to express Sox9 in a Cre recombinase-

To study the contribution of Sox9+ putative stem/progenitor cells beta-cell

Apr 1, 2010 . (A-D) Prx1-Cre and (E-H) Sox9-Cre cOE mutants. Robust alkaline phosphatase

In the developing skeleton, Sox9 was expressed before Runx2, an early

wt Sox9 cre/+. -13kb. - 6.5kb. 6.5 Kb wild type allele targeting vector. Sox9. Cre

Mar 26, 2011 . 3a, Supplementary Fig. 7) in which Cre-mediated recombination results in the

Apr 2, 2011 . However, XY Sf1:Cre(Tg/+); Sox9(flox/flox) mutant gonads develop as ovaries

skeletal progenitors using a Sox9-Cre knock-in allele. As Lmx1b is both .

Jul 3, 2008 . To test whether these early slow-cycling cells function as SCs, we use Sox9-Cre

Feb 15, 2009 . Generation of Taceflox/flox mice and Sox9-Cre knock-in mice was . The

Apr 15, 2009 . 5), Sox9-Cre (bone, cartilage, and various other tissue; ref. 6), Dermo1-Cre (

Jan 6, 2011 . Before discussing the role of Sox8 and Sox9 in ureter development in detail, we

Oct 11, 2005 . untranslated region of the Sox9 gene (Sox9-Cre knock-in). In the developing .

Here we report novel and different functions of Sox9 during mouse inner ear

Many of these genes and pathways including etv5, sox9, and fibroblast growth

We have therefore conditionally inactivated both Sox9 alleles in the gonadal

Oct 11, 2005 . Cell fate mapping by using Sox9-Cre;ROSA26 reporter (R26R) mice . By using

Cre-based lineage tracing showed that adult intestinal cells, hepatocytes and

In the developing skeleton, Sox9 was expressed before Runx2, an early osteoblast

By using Sox9-Cre;R26R mice, we also were able to systematically follow Sox9-

Nov 28, 2010 . (a) Experimental strategy of tamoxifen-inducible Cre-mediated cell tracking using

Oct 11, 2005 . Comparison of Sox9, Runx2, and Osx expression during embryonic development

In the developing skeleton, Sox9 wasexpressed before Runx2, an early

Feb 9, 2007 . To test whether these early slow-cycling cells function as SCs, we use Sox9-Cre

To study the regulation in vivo, SOX9 was ectopically expressed in hypertrophic

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